Archive for September, 2022

Disorders of Skin Pigmentation #skindisorders #skindisease #skinpigmentation

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Melanocytes are one of the cells of the epidermis. They produce the brown-black pigment called melanin which contributes to skin colour.
Albinism, hyperpigmentation, and hypopigmentation are some of the disorders of skin pigmentation. Vitiligo is a skin disorder caused by hypopigmentation of the epidermis due to the loss of melanin.

#pharmacy, #pharmacology, #clinicalpharmacology , #cellularandmolecularpharmacology, #pharmacotherapeutics, #biochemistry, #pharmacokinetics, #pharmacodynamics, #pathophysiology.
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7 Reasons You Might Be Sweating A Lot

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Chapters

0:00 Introduction
0:38 Runs in the family
1:02 Medication
1:34 Pregnancy
1:55 Thyroid Problems
2:24 Diabetes
2:52 Anxiety
3:10 Perimenopause

Hyperhidrosis is a condition characterized by abnormally increased sweating,[1] in excess of that required for regulation of body temperature.[2] Although primarily a physical burden, hyperhidrosis can deteriorate quality of life from a psychological, emotional, and social perspective.[3] This excess of sweat happens even if the person is not engaging in tasks that require muscular effort, and it does not depend on the exposure to heat.[4] Common places to sweat can include underarms, face, neck, back, groin, feet, and hands. It has been called by some ‘the silent handicap’.[5]

Both the words diaphoresis and hidrosis can mean either perspiration (in which sense they are synonymous with sweating[6][7]) or excessive perspiration, in which case they refer to a specific, narrowly defined, clinical disorder.
Hyperhidrosis can either be generalized, or localized to specific parts of the body. Hands, feet, armpits, groin, and the facial area are among the most active regions of perspiration due to the high number of sweat glands (eccrine glands in particular) in these areas. When excessive sweating is localized (e.g. palms, soles, face, underarms, scalp) it is referred to as primary hyperhidrosis or focal hyperhidrosis. Excessive sweating involving the whole body is termed generalized hyperhidrosis or secondary hyperhidrosis. It is usually the result of some other, underlying condition.

Primary or focal hyperhidrosis may be further divided by the area affected, for instance, palmoplantar hyperhidrosis (symptomatic sweating of only the hands or feet) or gustatory hyperhidrosis (sweating of the face or chest a few moments after eating certain foods).[1]

Hyperhidrosis can also be classified by onset, either congenital (present at birth) or acquired (beginning later in life). Primary or focal hyperhidrosis usually starts during adolescence or even earlier and seems to be inherited as an autosomal dominant genetic trait. It must be distinguished from secondary hyperhidrosis, which can start at any point in life. Secondary hyperhidrosis may be due to a disorder of the thyroid or pituitary glands, diabetes mellitus, tumors, gout, menopause, certain drugs, or mercury poisoning.[8]

One classification scheme uses the amount of skin affected.[9] In this scheme, excessive sweating in an area of 100 square centimeters (16 square inches) or more is differentiated from sweating that affects only a small area.[10]

Another classification scheme is based on possible causes of hyperhidrosis.
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View full lesson: https://ed.ted.com/lessons/why-do-we-sweat-john-murnan

There are a number of scenarios that can make us sweat–including exercise, eating spicy foods, and nervousness. But how does this substance suddenly materialize, and what exactly is its purpose? John Murnan explores the science behind sweat.

Lesson by John Murnan, directed by Dogzilla Studio.

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Vesiculobullous Skin Diseases | Pemphigus Vulgaris vs. Bullous Pemphigoid

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Vesiculobullous Skin Conditions

Lesson on Pemphigus vulgaris and Bullous Pemphigoid, differences and similarities between the two conditions, and diagnoses and treatments for both. Pemphigus vulgaris and Bullous Pemphigoid are autoimmune skin conditions characterized by bullae on the skin, but they differ greatly in their presentation and overall morbidity. In this lesson, you will learn about various clinical signs to distinguish these two conditions, as well as their differing pathophysiologies and treatments.

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JJ

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Check out some of my other lessons.

Medical Terminology – The Basics – Lesson 1:

Fatty Acid Synthesis Pathway:

Wnt/B Catenin Signaling Pathway:

Upper vs. Lower Motor Neuron Lesions:

Lesson on the Purine Synthesis and Salvage Pathway:

Gastrulation | Formation of Germ Layers:

Introductory lesson on Autophagy (Macroautophagy):

Infectious Disease Playlist

Dermatology Playlist

Pharmacology Playlist

Hematology Playlist

Rheumatology Playlist

Endocrinology Playlist

Nephrology Playlist

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**MEDICAL DISCLAIMER**: JJ Medicine does not provide medical advice, and the information available on this channel does not offer a diagnosis or advice regarding treatment. Information presented in these lessons is for educational purposes ONLY, and information presented here is not to be used as an alternative to a healthcare professional’s diagnosis and treatment of any person/animal.

Only a physician or other licensed healthcare professional are able to determine the requirement for medical assistance to be given to a patient. Please seek the advice of your physician or other licensed healthcare provider if you have any questions regarding a medical condition.

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*Although I try my best to present accurate information, there may be mistakes in this video. If you do see any mistakes with information in this lesson, please comment and let me know.*

I am always looking for ways to improve my lessons! Please don’t hesitate to leave me feedback and comments – all of your feedback is greatly appreciated! 🙂 And please don’t hesitate to send me any messages if you need any help – I will try my best to be here to help you guys 🙂

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JJ
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